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Daily Cardiology Research Analysis

3 papers

Three high-impact cardiology studies stood out. A multinational analysis showed an AI model applied to ECG images robustly predicts incident heart failure and improves risk stratification beyond established equations. A biomarker-driven subphenotyping framework for cardiogenic shock improved prognostication over SCAI shock staging and suggested treatment heterogeneity. A contemporary AMI in-hospital mortality model from 313,825 U.S. hospitalizations achieved excellent discrimination and bedside

Summary

Three high-impact cardiology studies stood out. A multinational analysis showed an AI model applied to ECG images robustly predicts incident heart failure and improves risk stratification beyond established equations. A biomarker-driven subphenotyping framework for cardiogenic shock improved prognostication over SCAI shock staging and suggested treatment heterogeneity. A contemporary AMI in-hospital mortality model from 313,825 U.S. hospitalizations achieved excellent discrimination and bedside usability.

Research Themes

  • AI-enabled cardiovascular risk prediction
  • Biomarker-driven shock phenotyping and precision cardiology
  • Contemporary outcome modeling and quality benchmarking in AMI

Selected Articles

1. Identifying biomarker-driven subphenotypes of cardiogenic shock: analysis of prospective cohorts and randomized controlled trials.

85Level IICohortEClinicalMedicine · 2025PMID: 39802301

Across two prospective cohorts, unsupervised clustering of plasma biomarkers revealed four reproducible cardiogenic shock subphenotypes. The inflammatory and cardiopathic classes had the highest 28-day mortality and improved risk stratification beyond SCAI stages. Applying a simplified classifier to three RCTs suggested potential heterogeneity of treatment effect by subphenotype.

Impact: This work operationalizes precision cardiology in shock by linking molecular phenotypes to outcomes and potential treatment heterogeneity, advancing beyond traditional hemodynamic staging.

Clinical Implications: Subphenotype assignment could refine prognostication, guide trial stratification/enrichment, and eventually inform phenotype-targeted therapies for cardiogenic shock.

Key Findings

  • Unsupervised clustering in two cohorts identified four biomarker-defined CS subphenotypes (adaptive, non-inflammatory, cardiopathic, inflammatory).
  • Inflammatory and cardiopathic subphenotypes had significantly higher 28-day mortality; adding subphenotype improved Harrell’s C-index beyond SCAI staging.
  • A simplified classifier assigned subphenotypes in three RCTs, enabling exploration of heterogeneity of treatment effect on 28-day mortality.

Methodological Strengths

  • Prospective cohorts with independent replication of clusters
  • Application and validation across multiple RCT datasets with a simplified classifier

Limitations

  • Biomarker panels and sampling times may differ between cohorts and trials
  • Observational nature of associations; not a randomized phenotype-guided intervention

Future Directions: Prospective, phenotype-guided trials to test tailored therapies; standardization of biomarker panels; integration with hemodynamics and imaging.

2. Heart failure risk stratification using artificial intelligence applied to electrocardiogram images: a multinational study.

82Level IICohortEuropean heart journal · 2025PMID: 39804243

An AI model applied to 12-lead ECG images predicted incident heart failure across three cohorts, with hazard ratios 3.9–23.5 and C-statistics up to 0.81. Adding AI-ECG to PCP-HF improved discrimination, supporting ECG-based digital biomarkers for HF risk stratification.

Impact: Demonstrates scalable, low-cost risk stratification using existing ECGs with AI, potentially enabling earlier HF prevention and more efficient population screening.

Clinical Implications: AI-ECG probability could triage patients for echocardiography, intensify risk modification, and augment PCP-HF in primary care and health systems.

Key Findings

  • AI-ECG positivity was associated with markedly higher incident HF risk across YNHHS, UKB, and ELSA-Brasil (HRs 3.88–23.50).
  • Discrimination ranged from 0.718 to 0.810; integrating AI-ECG with PCP-HF significantly improved risk prediction.
  • Risk increased monotonically with higher AI-ECG probabilities, robust to comorbidities and competing risk of death.

Methodological Strengths

  • Large, multinational external validation across three diverse cohorts
  • Comparison to guideline-based PCP-HF and demonstration of incremental predictive value

Limitations

  • Observational design limits causal inference about interventions triggered by AI-ECG
  • Model transportability to other healthcare systems and ECG acquisition workflows requires further testing

Future Directions: Prospective impact studies to test AI-ECG-triggered pathways, calibration in underrepresented groups, and integration with imaging and biomarkers.

3. Predicting Mortality in Patients Hospitalized With Acute Myocardial Infarction: From the National Cardiovascular Data Registry.

79Level IICohortCirculation. Cardiovascular quality and outcomes · 2025PMID: 39801472

Using 313,825 AMI hospitalizations across 784 U.S. sites, the authors developed and validated a 14-variable in-hospital mortality model (C-statistic 0.868) and a 0–25 bedside score. Out-of-hospital cardiac arrest, cardiogenic shock, and STEMI were the strongest predictors, and performance was consistent across subgroups and pandemic periods.

Impact: Provides an updated, validated benchmark for AMI care quality and bedside prognostication, enabling risk-standardized outcomes and informed clinical decisions.

Clinical Implications: Supports hospital benchmarking, triage, and shared decision-making; the simplified score facilitates quick risk assessment and resource allocation.

Key Findings

  • A 14-variable model achieved excellent discrimination (C-statistic 0.868) for AMI in-hospital mortality with good calibration.
  • Strongest predictors were out-of-hospital cardiac arrest, cardiogenic shock, and STEMI; a 0–25 point bedside score mapped to 0.3%–49.4% mortality risk.
  • Model performance was stable across MI type, hospital volume, and pre-/during COVID-19 periods.

Methodological Strengths

  • Very large, contemporary national registry with split-sample validation and bootstrapping
  • Parsimonious variable selection and creation of a user-friendly bedside score

Limitations

  • Registry-based observational data; potential residual confounding and coding variability
  • In-hospital outcome only; does not capture post-discharge events

Future Directions: Integration into EHRs for real-time risk dashboards; evaluation of impact on care pathways and outcomes; extension to post-discharge risk.