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Daily Report

Daily Cardiology Research Analysis

04/16/2025
3 papers selected
3 analyzed

Three impactful cardiology studies stood out today: (1) a Nature Communications mechanistic study linking dysregulated N-terminal protein acetylation (NAA10 variant) to arrhythmia and cardiomyopathy; (2) a global prospective registry in European Heart Journal clarifying anticoagulation risks in pregnancy with prosthetic valves, especially with LMWH and mitral mechanical valves; and (3) a JAMA Cardiology multi-cohort study showing AI can predict heart failure risk from single-lead ECGs, outperfor

Summary

Three impactful cardiology studies stood out today: (1) a Nature Communications mechanistic study linking dysregulated N-terminal protein acetylation (NAA10 variant) to arrhythmia and cardiomyopathy; (2) a global prospective registry in European Heart Journal clarifying anticoagulation risks in pregnancy with prosthetic valves, especially with LMWH and mitral mechanical valves; and (3) a JAMA Cardiology multi-cohort study showing AI can predict heart failure risk from single-lead ECGs, outperforming standard risk scores.

Research Themes

  • Mechanistic cardiogenetics: N-terminal acetylation and arrhythmia/cardiomyopathy
  • Anticoagulation strategies in pregnancy with prosthetic heart valves
  • AI-enabled cardiovascular risk stratification using wearable-scale ECG signals

Selected Articles

1. Dysregulation of N-terminal acetylation causes cardiac arrhythmia and cardiomyopathy.

88.5Level VBasic/Mechanistic Research
Nature communications · 2025PMID: 40235403

This mechanistic study identifies a previously unreported NAA10 p.(Arg4Ser) variant that segregates with QT prolongation, cardiomyopathy, and developmental delay in a large kindred, implicating dysregulated N-terminal acetylation as a disease mechanism. The findings link protein acetylation biology to human cardiac electrophysiology and structure, highlighting a new pathogenic pathway.

Impact: It uncovers a novel, potentially targetable molecular mechanism (N-terminal acetylation) underlying arrhythmia and cardiomyopathy, bridging human genetics with cardiac pathophysiology.

Clinical Implications: While early-stage, these findings support considering NAA10 and N-terminal acetylation pathways in genetic evaluation of arrhythmia/cardiomyopathy and motivate development of modulators of protein acetylation as future therapeutics.

Key Findings

  • A previously unidentified NAA10 p.(Arg4Ser) variant segregated with QT prolongation, cardiomyopathy, and developmental delay in a large kindred.
  • Dysregulation of N-terminal acetylation was implicated as a causal mechanism for cardiac arrhythmia and cardiomyopathy.
  • The study links protein N-terminal acetylation biology to human cardiac electrical and structural disease.

Methodological Strengths

  • Human genetic evidence with variant segregation in a large kindred
  • Mechanistic framing that connects post-translational modification to cardiac phenotype

Limitations

  • Limited sample centered on a single kindred; broader generalizability requires additional cohorts
  • Translational therapeutic implications remain to be tested in preclinical models

Future Directions: Validate NAA10-related acetylation defects across independent cohorts; delineate downstream substrates/pathways; assess therapeutic modulation of N-terminal acetylation in preclinical models.

N-terminal acetyltransferases including NAA10 catalyze N-terminal acetylation, an evolutionarily conserved co- and post-translational modification. However, little is known about the role of N-terminal acetylation in cardiac homeostasis. To gain insight into cardiac-dependent NAA10 function, we studied a previously unidentified NAA10 variant p.(Arg4Ser) segregating with QT-prolongation, cardiomyopathy, and developmental delay in a large kindred. Here, we show that the NAA10

2. Pregnancy with a prosthetic heart valve, thrombosis, and bleeding: the ESC EORP Registry of Pregnancy and Cardiac disease III.

79Level IICohort
European heart journal · 2025PMID: 40237423

In this global prospective registry of 613 pregnancies with prosthetic valves, biological valves had higher chances of uncomplicated live birth than mechanical valves (79% vs 54%). LMWH-based regimens were associated with more thromboembolic/hemorrhagic complications, valve thrombosis occurred in 6%, and mitral position strongly predicted thrombosis; anti-Xa monitoring benefits were inconclusive.

Impact: This is the most detailed contemporary prospective evidence to date informing anticoagulation choices and risk counseling in pregnant patients with prosthetic valves, with immediate implications for guideline updates.

Clinical Implications: For women anticipating pregnancy, biological valves yield more favorable outcomes. In mechanical valves, particularly in the mitral position, LMWH-based regimens appear to carry higher thromboembolic/bleeding risks; careful regimen selection, shared decision-making, and close monitoring are warranted.

Key Findings

  • Uncomplicated live birth was 54% with mechanical valves vs 79% with biological valves (P < .001).
  • LMWH-based regimens had the highest rates of thromboembolic and hemorrhagic complications; overall valve thrombosis occurred in 6%.
  • Mitral mechanical valve position predicted valve thrombosis (OR 3.3; 95% CI 1.9–8.0); anti-Xa monitoring benefits on events were inconclusive (P=0.060).

Methodological Strengths

  • Prospective, global registry with detailed anticoagulation dosing and monitoring data
  • Large sample capturing mechanical and biological valve pregnancies with clinically relevant endpoints

Limitations

  • Observational design with potential confounding by indication; not randomized
  • Anti-Xa monitoring analysis underpowered for definitive benefit assessment

Future Directions: Randomized or carefully controlled comparative studies of anticoagulation strategies in mechanical valve pregnancy, with standardized anti-Xa protocols and valve-position–specific risk stratification.

BACKGROUND AND AIMS: Pregnancy in women with a prosthetic heart valve is considered high risk, primarily due to the need for effective anticoagulation. However, data on the relationship between anticoagulation practices and pregnancy outcomes are very limited. METHODS: The Registry of Pregnancy and Cardiac disease is a global registry that prospectively enrolled pregnancies in women with a prosthetic heart valve between January 2018 and April 2023. Detailed data on anticoagulation, including dosage and monitoring, and cardiovascular, pregnancy, and perinatal outcomes were collected. RESULTS: In total, 613 pregnancies were included of which 411 pregnancies were in women with a mechanical valve and 202 were in women with a biological valve. The chance of an uncomplicated pregnancy with a live birth in women with a mechanical valve was 54%, compared with 79% in women with a biological valve (P < .001). Thromboembolic and haemorrhagic complications most frequently occurred when low-molecular weight heparin (LMWH)-based regimens were used. Valve thrombosis occurred in 24 (6%) women, and a prosthetic valve in mitral position was associated with valve thrombosis (odds ratio 3.3; 95% confidence interval 1.9-8.0). A thromboembolic event occurred in 12 (10%) women with anti-Xa monitoring and in 9 (21%) women without (P = .060). Foetal death occurred in 20% of all pregnancies. CONCLUSIONS: More favourable outcomes were found in women with a biological valve compared with a mechanical valve. In women with a mechanical valve, the use of LMWH was associated with an increased risk of thromboembolic complications. A mitral prosthetic valve was identified as a predictor for valve thrombosis. The benefit could not be confirmed nor refuted, in terms of reduced thromboembolic events, from using anti-Xa level monitoring in women on LMWH.

3. Artificial Intelligence-Enabled Prediction of Heart Failure Risk From Single-Lead Electrocardiograms.

77Level IICohort
JAMA cardiology · 2025PMID: 40238120

A noise-adapted AI model using lead I ECGs predicted new-onset HF across three multinational cohorts (N≈248,000), with C-statistics 0.72–0.83 and substantial improvements over PCP-HF and PREVENT scores (C-statistic gains ~0.07–0.11; IDI 0.068–0.205; NRI up to ~47%). Each 0.1 increase in model probability conferred 27–65% higher hazard, independent of clinical covariates.

Impact: Demonstrates scalable HF risk prediction from single-lead ECG signals compatible with wearables, outperforming established risk scores and enabling community-level screening strategies.

Clinical Implications: AI-ECG could be integrated into primary care and wearable platforms to flag high-risk individuals for echocardiography/biomarker testing, earlier initiation of guideline-directed HF prevention (e.g., SGLT2 inhibitors), and targeted lifestyle interventions.

Key Findings

  • Across YNHHS, UK Biobank, and ELSA-Brasil, AI-ECG discrimination for new-onset HF was 0.723, 0.736, and 0.828, respectively.
  • Each 0.1 increase in model probability associated with a 27–65% higher hazard of incident HF independent of confounders.
  • AI-ECG improved risk prediction beyond PCP-HF and PREVENT (C-statistic gains 0.069–0.107; IDI 0.068–0.205; NRI 11.8–47.5%).

Methodological Strengths

  • Large, multi-cohort external validation across healthcare and population cohorts
  • Noise-adapted modeling reflecting wearable ECG quality; head-to-head comparison with standard HF risk equations

Limitations

  • Retrospective design; prospective implementation studies are needed to assess real-world workflow and outcomes impact
  • Outcome limited to first HF hospitalization; subclinical HF and outpatient events not captured

Future Directions: Prospective trials embedding AI-ECG screening in wearable and primary care workflows to test downstream testing, treatment initiation, patient-reported outcomes, and cost-effectiveness.

IMPORTANCE: Despite the availability of disease-modifying therapies, scalable strategies for heart failure (HF) risk stratification remain elusive. Portable devices capable of recording single-lead electrocardiograms (ECGs) may enable large-scale community-based risk assessment. OBJECTIVE: To evaluate whether an artificial intelligence (AI) algorithm can predict HF risk from noisy single-lead ECGs. DESIGN, SETTING, AND PARTICIPANTS: A retrospective cohort study of individuals without HF at baseline was conducted among individuals with conventionally obtained outpatient ECGs in the integrated Yale New Haven Health System (YNHHS) and prospective population-based cohorts of the UK Biobank (UKB) and the Brazilian Longitudinal Study of Adult Health (ELSA-Brasil). Data analysis was performed from September 2023 to February 2025. EXPOSURE: AI-ECG-defined risk of left ventricular systolic dysfunction (LVSD). MAIN OUTCOMES AND MEASURES: Among individuals with ECGs, lead I ECGs were isolated and a noise-adapted AI-ECG model (to simulate ECG signals from wearable devices) trained to identify LVSD was deployed. The association of the model probability with new-onset HF, defined as the first HF hospitalization, was evaluated. The discrimination of AI-ECG was compared against 2 risk scores for new-onset HF (Pooled Cohort Equations to Prevent Heart Failure [PCP-HF] and Predicting Risk of Cardiovascular Disease Events [PREVENT] equations) using the Harrel C statistic, integrated discrimination improvement, and net reclassification improvement. RESULTS: There were 192 667 YNHHS patients (median [IQR] age, 56 [41-69] years; 111 181 women [57.7%]), 42 141 UKB participants (median [IQR] age, 65 [59-71] years; 21 795 women [51.7%]), and 13 454 ELSA-Brasil participants (median [IQR] age, 51 [45-58] years; 7348 women [54.6%]) with baseline ECGs. A total of 3697 (1.9%) developed HF in YNHHS over a median (IQR) of 4.6 (2.8-6.6) years, 46 (0.1%) in UKB over a median (IQR) of 3.1 (2.1-4.5) years, and 31 (0.2%) in ELSA-Brasil over a median (IQR) of 4.2 (3.7-4.5) years. A positive AI-ECG screening result for LVSD was associated with a 3- to 7-fold higher risk for HF, and each 0.1 increment in the model probability was associated with a 27% to 65% higher hazard across cohorts, independent of age, sex, comorbidities, and competing risk of death. AI-ECG's discrimination for new-onset HF was 0.723 (95% CI, 0.694-0.752) in YNHHS, 0.736 (95% CI, 0.606-0.867) in UKB, and 0.828 (95% CI, 0.692-0.964) in ELSA-Brasil. Across cohorts, incorporating AI-ECG predictions alongside PCP-HF and PREVENT equations was associated with a higher Harrel C statistic (difference in addition to PCP-HF, 0.080-0.107; difference in addition to PREVENT, 0.069-0.094). AI-ECG had an integrated discrimination improvement of 0.091 to 0.205 vs PCP-HF and 0.068 to 0.192 vs PREVENT; it had a net reclassification improvement of 18.2% to 47.2% vs PCP-HF and 11.8% to 47.5% vs PREVENT. CONCLUSIONS AND RELEVANCE: Across multinational cohorts, a noise-adapted AI-ECG model estimated HF risk using lead I ECGs, suggesting a potential HF risk-stratification strategy requiring prospective study using wearable and portable ECG devices.