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Daily Cardiology Research Analysis

3 papers

Three studies stand out today: a JCI mechanistic paper identifies a smooth muscle lncRNA (CARMN) as a regulator of angiogenesis via the miR-143-3p–HHIP–Hedgehog axis in chronic limb-threatening ischemia; a national TVT Registry analysis suggests balloon-expandable valve-in-valve TAVR has favorable 5-year outcomes compared with native TAVR; and a multicenter cohort shows that preoperative NT-proBNP reduction predicts better 30-day and 5-year outcomes after cardiac surgery.

Summary

Three studies stand out today: a JCI mechanistic paper identifies a smooth muscle lncRNA (CARMN) as a regulator of angiogenesis via the miR-143-3p–HHIP–Hedgehog axis in chronic limb-threatening ischemia; a national TVT Registry analysis suggests balloon-expandable valve-in-valve TAVR has favorable 5-year outcomes compared with native TAVR; and a multicenter cohort shows that preoperative NT-proBNP reduction predicts better 30-day and 5-year outcomes after cardiac surgery.

Research Themes

  • Translational mechanisms and targets in peripheral artery disease
  • Real-world outcomes of structural heart interventions
  • Biomarker-guided perioperative risk optimization

Selected Articles

1. A smooth muscle cell lncRNA controls angiogenesis in chronic limb-threatening ischemia through miR-143-3p/HHIP signaling.

76Level VCase-controlThe Journal of clinical investigation · 2025PMID: 40875440

This mechanistic study identifies CARMN as a smooth muscle-enriched lncRNA that promotes angiogenesis via a miR-143-3p–HHIP–Hedgehog axis in limb ischemia. Loss of CARMN impairs capillary formation and perfusion, whereas miR-143-3p mimics or HHIP silencing rescue endothelial defects and restore blood flow.

Impact: It reveals a tractable SMC–EC signaling axis for therapeutic angiogenesis in CLTI, highlighting miR-143-3p/HHIP as targets with in vivo rescue evidence.

Clinical Implications: Therapies enhancing miR-143-3p or inhibiting HHIP could augment angiogenesis and perfusion in CLTI. It supports cell–cell crosstalk–based strategies beyond VEGF-centric approaches.

Key Findings

  • CARMN is downregulated in human CLTI muscle and is SMC-enriched.
  • CARMN knockout impairs capillary density and blood flow recovery after limb ischemia.
  • CARMN regulates Hedgehog signaling via miR-143-3p targeting HHIP; miR-143-3p or HHIP siRNA rescues endothelial angiogenic defects and perfusion.

Methodological Strengths

  • Multimodal validation including knockout mice, endothelial functional assays, and RNA-seq pathway analysis
  • In vivo rescue experiments (miR-143-3p mimic, HHIP siRNA) demonstrating causality

Limitations

  • Preclinical models; human interventional data are lacking
  • Delivery, specificity, and durability of miRNA/siRNA therapies remain to be established

Future Directions: Develop targeted delivery systems for miR-143-3p/HHIP modulation, validate efficacy and safety in large-animal models, and progress to early-phase trials in CLTI.

2. Mid-Term Outcomes of Balloon-Expandable Aortic Valve-in-Valve Replacement in the United States.

73Level IIICohortJACC. Cardiovascular interventions · 2025PMID: 40866029

In a propensity-matched TVT Registry analysis (13,638 pairs), balloon-expandable valve-in-valve TAVR showed lower 5-year death and stroke compared with native TAVR. Hemodynamics were favorable, particularly with SAPIEN 3 Ultra RESILIA, supporting the safety and durability of AViV across risk strata.

Impact: Largest real-world, mid-term comparison indicates AViV may confer survival and cerebrovascular advantages over native TAVR, informing structural heart strategies in failed surgical bioprostheses.

Clinical Implications: For degenerated surgical valves, AViV with BEVs appears safe and durable at 5 years and may be favored over native TAVR in appropriate anatomy; device selection (e.g., RESILIA) affects gradients.

Key Findings

  • Propensity-matched analysis (13,638 pairs) showed lower 5-year death (43.1% vs 55.2%) and stroke (10.5% vs 11.8%) with AViV vs native TAVR.
  • SAPIEN 3 Ultra RESILIA had lower discharge gradients across all valve sizes compared with predecessors.
  • Outcomes were consistent across STS risk tertiles; no 5-year death/stroke difference between stented and stentless prior surgical valves.

Methodological Strengths

  • Very large national registry with propensity matching and 5-year follow-up
  • Subgroup analyses by risk, device subtype, and surgical valve type

Limitations

  • Observational design with residual confounding despite matching
  • Heterogeneity in centers, devices, and evolving practice over inclusion period

Future Directions: Prospective comparative studies of AViV strategies, optimization of device sizing/BEV choice, and durability assessments beyond 5 years.

3. Prognostic Implications of Preoperative N-Terminal Pro-B-Type Natriuretic Peptide Dynamics in Patients Undergoing Cardiac Surgery.

71.5Level IICohortJACC. Advances · 2025PMID: 40865190

In 6,938 consecutive cardiac surgery patients with external validation, lower preoperative NT-proBNP and reductions from diagnostic levels were independently associated with lower 30-day and 5-year mortality and less resource use. Optimizing patients to reduce NT-proBNP before surgery may improve outcomes.

Impact: Links a modifiable biomarker trajectory to perioperative and long-term outcomes, providing an actionable target for prehabilitation and risk optimization.

Clinical Implications: Incorporate NT-proBNP dynamics into preoperative assessment; consider optimization strategies (e.g., decongestion, GDMT titration) to lower NT-proBNP prior to surgery to reduce mortality and resource use.

Key Findings

  • High preoperative NT-proBNP predicted higher 30-day and 5-year mortality after cardiac surgery.
  • Patients whose high NT-proBNP decreased below 3,000 ng/L before surgery had lower 30-day mortality (HR 0.21) and improved 5-year survival.
  • NT-proBNP reduction associated with shorter ICU stay and less ultrafiltration and ECMO use; findings validated in SWEDEHEART.

Methodological Strengths

  • Large consecutive cohort with EuroSCORE II–adjusted models and external validation
  • Trajectory analysis of biomarker dynamics rather than single-point measurement

Limitations

  • Observational design cannot prove causality; unmeasured confounding possible
  • Specific optimization interventions to lower NT-proBNP were not tested

Future Directions: Randomized trials testing preoperative optimization strategies targeting NT-proBNP reduction and integration into perioperative risk algorithms.