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Daily Endocrinology Research Analysis

3 papers

Three studies advance endocrine diagnostics and risk stratification: a prospective modification of the overnight dexamethasone suppression test using late-afternoon cortisol and ACTH improved diagnostic accuracy for Cushing syndrome; a large real-world analysis shows 24-hour urinary aldosterone retains strong performance for diagnosing primary aldosteronism even without antihypertensive washout; and a nationwide cohort links so-called non-functional adrenal tumors to substantially higher risks o

Summary

Three studies advance endocrine diagnostics and risk stratification: a prospective modification of the overnight dexamethasone suppression test using late-afternoon cortisol and ACTH improved diagnostic accuracy for Cushing syndrome; a large real-world analysis shows 24-hour urinary aldosterone retains strong performance for diagnosing primary aldosteronism even without antihypertensive washout; and a nationwide cohort links so-called non-functional adrenal tumors to substantially higher risks of psychiatric and sleep disorders.

Research Themes

  • Endocrine diagnostic optimization
  • Adrenal disorders and cardiometabolic risk
  • Neuropsychiatric comorbidity in adrenal incidentalomas

Selected Articles

1. Overnight Dexamethasone Suppression Test: Enhanced Accuracy with Late-Afternoon Cortisol and Morning/Late-Afternoon ACTH.

77Level IICohortThe Journal of clinical endocrinology and metabolism · 2025PMID: 40972562

In a prospective diagnostic study, adding late-afternoon cortisol and ACTH to the standard 1‑mg ODST improved specificity, particularly in COCP and CKD, without sacrificing sensitivity for ACTH-dependent Cushing syndrome. A 4‑PM cortisol and 8‑AM/4‑PM ACTH (10 pg/mL cut-off) achieved excellent discrimination, and 4‑PM cortisol differentiated remission from persistent Cushing disease with 98.1% accuracy.

Impact: Offers a practical, immediately applicable enhancement to a widely used endocrine test, addressing known specificity issues in challenging subgroups. Could standardize a refined ODST protocol.

Clinical Implications: Consider measuring 4‑PM cortisol and ACTH alongside standard ODST sampling, especially in COCP and CKD, to improve specificity and postoperative monitoring of Cushing disease.

Key Findings

  • 4‑PM cortisol was significantly lower than 8‑AM in healthy, COCP, CKD, and CD remission groups, but not in untreated ACTH-dependent Cushing syndrome or persistent Cushing disease.
  • Using a 10 pg/mL ACTH cut-off at 8‑AM and 4‑PM yielded high specificity in healthy (95–100%), COCP (100%), and CKD (96.6–100%) with 100% sensitivity for A‑CS.
  • 4‑PM cortisol discriminated Cushing disease remission vs persistence with 98.1% accuracy, outperforming morning ACTH.

Methodological Strengths

  • Prospective, protocolized sampling with multiple clinically relevant subgroups (COCP, CKD, remission/persistent CD).
  • Use of prespecified cut-offs and comparison of morning vs late‑afternoon measures with clear performance metrics.

Limitations

  • Single-center design with modest subgroup sizes; external validation is needed.
  • Potential assay and laboratory variability may affect generalizability of exact cut-offs.

Future Directions: Multicenter validation of 4‑PM cortisol/ACTH thresholds and cost-effectiveness analyses; integration into diagnostic algorithms and postoperative surveillance protocols.

2. Diagnostic performance of 24-hour urinary aldosterone for primary aldosteronism in patients with or without discontinuation of antihypertensive medications.

74.5Level IIICohortHypertension research : official journal of the Japanese Society of Hypertension · 2025PMID: 40973763

In 842 derivation patients plus 157 external validations, 24‑h urinary aldosterone discriminated PA from essential hypertension with similar AUCs whether antihypertensives were stopped or continued. Optimal cut-offs were 9.57 μg off medications and 8.41 μg on medications, supporting feasible diagnosis without washout if thresholds are adjusted.

Impact: Addresses a major practical barrier in PA diagnosis—medication washout—by demonstrating robust performance of 24‑h Uald under real-world conditions and providing adjusted cut-offs.

Clinical Implications: Clinicians can consider 24‑h Uald for PA screening/confirmation without drug washout, using lower thresholds when patients remain on antihypertensives; unilateral PA tends to show higher Uald.

Key Findings

  • AUCs for diagnosing PA were similar off vs on medications (0.879 vs 0.851).
  • Optimal 24‑h Uald cut-offs: 9.57 μg (off meds; sensitivity 79.4%, specificity 88.6%) and 8.41 μg (on meds; sensitivity 75.8%, specificity 79.4%).
  • External validation accuracy reached 89.2%; combination antihypertensive therapy lowered Uald, requiring adjusted thresholds.
  • Unilateral PA exhibited higher 24‑h Uald levels than bilateral PA.

Methodological Strengths

  • Large derivation cohort with an external validation set.
  • Stratified analyses by antihypertensive regimens and PA lateralization.

Limitations

  • Retrospective design with heterogeneous medication exposures and non-standardized dietary sodium.
  • Single-country setting may limit generalizability of exact thresholds.

Future Directions: Prospective, multiethnic validation of on‑medication cut-offs and integration with ARR/imaging to streamline PA pathways.

3. Psychiatric and Sleep disorders in Patients with Non-Functional Adrenal Tumors.

74.5Level IIICohortThe Journal of clinical endocrinology and metabolism · 2025PMID: 40973669

In a nationwide register-based cohort of 17,561 NFAT patients with 5.4 years median follow-up, both prior prevalence and incident risk of psychiatric and/or sleep disorders were nearly doubled compared with controls, consistent across secondary outcomes and sensitivity analyses.

Impact: Challenges the notion of ‘non-functional’ adrenal incidentalomas by documenting substantial neuropsychiatric and sleep morbidity, prompting reconsideration of evaluation and follow-up strategies.

Clinical Implications: NFAT patients may warrant proactive screening and management for psychiatric and sleep disorders, and reassessment for mild autonomous cortisol secretion in those with new-onset symptoms.

Key Findings

  • Adjusted odds of prior psychiatric/sleep disorders were approximately doubled in NFAT vs controls (adjusted OR 2.06, 95% CI 1.98–2.14).
  • Incident psychiatric/sleep disorders were higher in NFAT during follow-up (adjusted HR 1.92, 95% CI 1.82–2.01; median follow-up 5.4 years).
  • Elevations were consistent across secondary outcomes (e.g., mood, anxiety/stress-related, psychotic, substance use disorders) and multiple sensitivity analyses.

Methodological Strengths

  • Very large, nationwide cohort with extensive adjustment for demographics and socioeconomic factors.
  • Multiple sensitivity analyses and consistent findings across secondary outcomes.

Limitations

  • Retrospective registry data may be subject to residual confounding and diagnostic misclassification.
  • Lack of biochemical cortisol profiling limits mechanistic inference regarding mild cortisol excess.

Future Directions: Prospective studies integrating biochemical cortisol assessment and interventional trials to test whether addressing MACS reduces neuropsychiatric burden.