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Daily Respiratory Research Analysis

3 papers

Three impactful studies reshaped respiratory medicine today: a Thorax analysis found cardiopulmonary exercise testing (CPET) does not improve prediction of post-lung resection complications beyond routine data; a RECOVER-EHR study in The Lancet Infectious Diseases showed pediatric SARS-CoV-2 reinfection doubles risk of long COVID and multiple sequelae; and a Mendelian randomization plus clinical study linked gut microbiota (notably Akkermansia) to pneumonia outcomes, supporting a causal gut–lung

Summary

Three impactful studies reshaped respiratory medicine today: a Thorax analysis found cardiopulmonary exercise testing (CPET) does not improve prediction of post-lung resection complications beyond routine data; a RECOVER-EHR study in The Lancet Infectious Diseases showed pediatric SARS-CoV-2 reinfection doubles risk of long COVID and multiple sequelae; and a Mendelian randomization plus clinical study linked gut microbiota (notably Akkermansia) to pneumonia outcomes, supporting a causal gut–lung axis.

Research Themes

  • De-implementation of low-value preoperative testing in thoracic surgery
  • Pediatric long COVID risk after SARS-CoV-2 reinfection
  • Causal gut–lung axis in pneumonia via Mendelian randomization

Selected Articles

1. Cardiopulmonary exercise testing before lung resection surgery: still indicated? Evaluating predictive utility using machine learning.

76Level IIICohortThorax · 2025PMID: 41043965

Across two prospective multicenter cohorts (n=497), adding CPET variables did not improve machine-learning prediction of postoperative pulmonary or cardiovascular complications beyond preoperative PFTs and clinical data. Findings were consistent in unselected candidates and those meeting ACCP or ERS/ESTS CPET criteria.

Impact: This challenges entrenched preoperative testing paradigms and supports de-implementation of routine CPET when baseline data suffice, potentially reducing costs and patient burden.

Clinical Implications: Consider limiting CPET to select scenarios (e.g., equivocal PFTs, unexplained dyspnea) rather than routine use in lung resection candidates, and update risk stratification guidelines accordingly.

Key Findings

  • CPET did not improve prediction of postoperative pulmonary complications (AUC-ROC 0.72–0.78 without benefit; p=0.47).
  • CPET did not improve prediction of postoperative cardiovascular complications (AUC-ROC 0.65–0.73; p=0.96).
  • Results were consistent in subgroups meeting ACCP or ERS/ESTS CPET criteria (no performance gain).

Methodological Strengths

  • Prospective multicenter cohorts with standardized preoperative assessments
  • Nested cross-validation across multiple machine-learning algorithms with subgroup analyses (ACCP, ERS/ESTS)

Limitations

  • Secondary analysis without external validation cohort
  • In-hospital outcomes only; long-term complications not assessed

Future Directions: Prospective pragmatic trials to test de-implementation of routine CPET and to identify specific clinical contexts where CPET truly adds value.

2. Long COVID associated with SARS-CoV-2 reinfection among children and adolescents in the omicron era (RECOVER-EHR): a retrospective cohort study.

74.5Level IIICohortThe Lancet. Infectious diseases · 2025PMID: 41043442

In 465,717 children/adolescents across 40 US institutions, SARS-CoV-2 reinfection (12.5%) during the omicron era doubled the risk of clinician-coded PASC (RR 2.08) compared with the first infection, and increased risks across multiple organ systems. Findings support prevention (e.g., vaccination) and targeted follow-up after reinfection.

Impact: Provides high-quality, large-scale evidence that pediatric reinfection substantially elevates long-COVID risk, informing vaccination and surveillance strategies.

Clinical Implications: Prioritize vaccination and boosters in children/adolescents and implement post-reinfection monitoring for PASC symptoms across organ systems.

Key Findings

  • Reinfection doubled the risk of clinician-coded PASC (U09.9) versus first infection (RR 2.08, 95% CI 1.68–2.59).
  • Increased risks spanned cardiovascular, renal, neurologic, autonomic (POTS/dysautonomia), hepatic, respiratory, and mental health domains (RR 1.15–3.60).
  • Incidence of PASC diagnoses per million per 6 months: 903.7 for first infection vs 1883.7 for reinfection.

Methodological Strengths

  • Large, multicenter cohort with propensity score and exact matching to mitigate confounding
  • Comprehensive evaluation of multisystem PASC endpoints with standardized coding (U09.9)

Limitations

  • Retrospective EHR data prone to misclassification and variable coding practices
  • Residual confounding and healthcare-seeking behavior differences cannot be fully excluded

Future Directions: Prospective cohorts to validate reinfection-associated PASC risk, define high-risk phenotypes, and evaluate vaccine/antiviral strategies to mitigate long-term sequelae.

3. Causal relationship between gut microbiota and pneumonia: a Mendelian randomization and retrospective case-control study.

70Level IIICase-controlBMC pulmonary medicine · 2025PMID: 41044486

Integrating Mendelian randomization with ICU case-control microbiome profiling, the study identified potential causal links between gut taxa and pneumonia outcomes. Akkermansia showed a protective association, including a lower 28-day critical-care mortality (OR 0.42) and correlations with reduced lactate and ICU stay in septic ARDS.

Impact: Provides causal inference supporting the gut–lung axis in pneumonia and nominates Akkermansia as a potential therapeutic target or biomarker in critical illness.

Clinical Implications: Motivates trials of microbiome-modulating interventions (e.g., pre/probiotics or dietary strategies) and monitoring of gut taxa such as Akkermansia in septic respiratory failure.

Key Findings

  • MR identified multiple gut taxa with potential causal effects on critical-care pneumonia and 28-day mortality.
  • Akkermansia was associated with reduced 28-day death in critical-care pneumonia (OR 0.42, 95% CI 0.22–0.79, p=0.007).
  • In septic ARDS, Akkermansia levels correlated negatively with lactate and ICU length of stay.

Methodological Strengths

  • Two-sample MR with multiple estimators (IVW, weighted median, MR-Egger) and pleiotropy checks (MR-PRESSO, Egger intercept)
  • Independent ICU case-control validation with 16S rRNA sequencing and PERMANOVA

Limitations

  • MR relies on validity of genetic instruments and may be affected by residual pleiotropy or weak instruments
  • Single-center ICU microbiome sample with limited size; generalizability requires replication

Future Directions: Interventional studies modulating Akkermansia and broader microbiota to test causality, and multi-center validation of microbiome–pneumonia associations.