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Daily Anesthesiology Research Analysis

3 papers

Three impactful anesthesiology-related studies span basic science, neuroprognostication, and perioperative outcomes. A mechanistic study uncovers a novel XPO1–HMGB1 pathway driving morphine tolerance with therapeutic potential. Continuous EEG assessed early and late improves outcome prediction after cardiac arrest without false positives, while a 14,129-patient cohort links preoperative sleep disturbance to wide-ranging postoperative adverse outcomes.

Summary

Three impactful anesthesiology-related studies span basic science, neuroprognostication, and perioperative outcomes. A mechanistic study uncovers a novel XPO1–HMGB1 pathway driving morphine tolerance with therapeutic potential. Continuous EEG assessed early and late improves outcome prediction after cardiac arrest without false positives, while a 14,129-patient cohort links preoperative sleep disturbance to wide-ranging postoperative adverse outcomes.

Research Themes

  • Mechanisms of opioid tolerance and potential therapeutic targets
  • Continuous EEG prognostication after cardiac arrest
  • Impact of preoperative sleep disturbance on perioperative outcomes

Selected Articles

1. Parylation dependent nuclear export of HMGB1 via exportin 1 contributes to morphine tolerance.

76Level VCase-controlBrain, behavior, and immunity · 2025PMID: 40782988

In a rat model, morphine tolerance was driven by STK38-mediated phosphorylation of XPO1 and PARP1-dependent PARylation of HMGB1, enabling XPO1-dependent nuclear export and extracellular release of HMGB1. Pharmacologic co-inhibition of XPO1 and PARP1 reversed established morphine tolerance and mechanical hypersensitivity, whereas intrathecal HMGB1 negated these benefits.

Impact: This work identifies a previously unrecognized XPO1–HMGB1 axis as a mechanistic driver of opioid tolerance and demonstrates a tractable combinatorial therapy to reverse it. It reframes nuclear export as a therapeutic node in analgesic tolerance.

Clinical Implications: Although preclinical, targeting nuclear export (XPO1) and PARP1 could augment perioperative opioid analgesia, limit dose escalation, and reduce opioid-related adverse effects. Translation will require safety, CNS-penetration, and selectivity assessments.

Key Findings

  • Chronic morphine upregulated spinal XPO1 and induced STK38-dependent phosphorylation at Ser1010, enhancing nuclear export function.
  • CSF proteomics showed elevated HMGB1 after morphine; XPO1 inhibition suppressed HMGB1 secretion.
  • PARP1-mediated PARylation of HMGB1 was required for its interaction with XPO1 and nuclear export.
  • Combined low-dose inhibition of XPO1 and PARP1 reversed morphine tolerance and mechanical hypersensitivity; intrathecal HMGB1 abolished these effects.

Methodological Strengths

  • Multimodal approach integrating spinal biochemistry, CSF proteomics, protein–protein interaction assays, and behavioral phenotyping.
  • Mechanistic causality supported by kinase mapping (STK38), PARP1 modulation, and pharmacologic co-inhibition with behavioral rescue.

Limitations

  • Preclinical animal study without human validation; translational relevance and dosing/safety remain unknown.
  • Potential off-target effects of XPO1/PARP1 inhibitors and systemic immunomodulation require careful evaluation.

Future Directions: Validate the XPO1–HMGB1 pathway in human tissues/CSF from opioid-tolerant patients; test CNS-selective XPO1/PARP1 modulation in large animals; assess safety, dosing, and potential synergy with multimodal analgesia.

2. Assessing both early and late EEG patterns improves prediction of outcome after cardiac arrest.

71.5Level IICohortResuscitation · 2025PMID: 40783100

In comatose post–cardiac arrest patients from the TTM2 cohort, early (≤24 h) and late (>24 h) highly malignant EEG patterns each achieved 100% specificity for poor outcome, but limited sensitivity. Combining early and late predictors and adding cEEG information over 36 h increased sensitivity to 49% without false positives; a continuous background within 12 h predicted good outcome.

Impact: Provides a pragmatic, time-structured cEEG strategy that improves sensitivity while maintaining zero false positives in this cohort, directly informing neuroprognostication bundles.

Clinical Implications: Incorporate continuous EEG during the first 36 h post–cardiac arrest; actively search for early and late highly malignant patterns while recognizing continuous background early as a favorable sign. Use within multimodal prognostication to guide timing of decisions.

Key Findings

  • Early EEG predictors (≤24 h) had 100% specificity and up to 30% sensitivity for poor outcome.
  • Late EEG predictors (>24 h) had 100% specificity and up to 32% sensitivity.
  • Combining early and late cEEG predictors across time improved sensitivity to 49% by 36 h without false positive survivors (p = 0.001).
  • A continuous EEG background within 12 h predicted good outcome (sensitivity 61%, specificity 87%).

Methodological Strengths

  • Blinded cEEG assessment using standardized ACNS terminology within a contemporary randomized trial cohort (TTM2).
  • Predefined early and late EEG categories aligned with European post-resuscitation guidelines.

Limitations

  • Observational design with potential self-fulfilling prophecy bias due to withdrawal-of-care influenced by EEG findings.
  • Modest sample size (n=191) limits precision of sensitivity estimates and subgroup analyses.

Future Directions: Prospective multicenter validation with standardized treatment-limitation policies; integrate quantitative EEG metrics and multimodal biomarkers to further increase sensitivity without compromising specificity.

3. Associations of preoperative sleep disturbance with intraoperative and postoperative adverse outcomes among Chinese surgical patients: evidence from the China surgery and anesthesia cohort (CSAC).

71Level IICohortJournal of clinical anesthesia · 2025PMID: 40782427

In 14,129 surgical patients aged 40–65 years, preoperative sleep disturbance (PSQI) was associated with increased risks of two in-hospital and all eight post-discharge adverse outcomes. The highest risks occurred in patients with multiple sleep problems and daytime dysfunction, and mediation analyses implicated postoperative sleep disturbance and depression as mediators of long-term adverse outcomes.

Impact: Large, contemporary, multicenter evidence links preoperative sleep disturbance to a broad spectrum of adverse outcomes and elucidates mediating pathways, highlighting a modifiable perioperative risk factor.

Clinical Implications: Implement routine preoperative sleep screening (e.g., PSQI), stratify high-risk 'multiple problems/daytime dysfunction' phenotypes, and deploy sleep optimization and mental health interventions pre- and postoperatively to mitigate long-term adverse outcomes.

Key Findings

  • Among 14,129 patients (mean age 52.3 years; 58.8% female), preoperative sleep disturbance was associated with two in-hospital and all eight post-discharge adverse outcomes.
  • Strongest associations were for postoperative moderate-to-severe sleep disturbance (ORs 3.88–18.64 at 1 month; 3.44–13.31 at 6 months; 3.98–15.58 at 12 months) and depression (ORs 1.88–5.60 across timepoints).
  • K-means clusters identified a 'multiple sleep problems with daytime dysfunction' phenotype with the highest risk across outcomes.
  • Mediation analyses indicated postoperative sleep disturbance and depression significantly mediated long-term adverse outcomes.

Methodological Strengths

  • Large multicenter cohort with comprehensive perioperative outcome capture across acute and long-term timepoints.
  • Use of clustering to define sleep phenotypes and mediation analysis to probe mechanistic pathways.

Limitations

  • Observational design with potential residual confounding; sleep quality assessed by self-report (PSQI).
  • Age range limited to 40–65 years; generalizability to older or younger populations is uncertain.

Future Directions: Randomized trials testing perioperative sleep optimization and mental health interventions in high-risk phenotypes; integration of objective sleep measures (actigraphy/polysomnography) and biological markers.