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Daily Anesthesiology Research Analysis

3 papers

Three impactful studies shape perioperative and critical care practice. A translational study identifies suPAR as a kidney-specific vasoconstrictor linking innate immunity to perioperative AKI risk. Two large perioperative cohorts show that FIB-4 liver fibrosis scores and preoperative HbA1c (including undiagnosed diabetes) independently predict 30-day postoperative complications and mortality, supporting biomarker-driven risk stratification and screening.

Summary

Three impactful studies shape perioperative and critical care practice. A translational study identifies suPAR as a kidney-specific vasoconstrictor linking innate immunity to perioperative AKI risk. Two large perioperative cohorts show that FIB-4 liver fibrosis scores and preoperative HbA1c (including undiagnosed diabetes) independently predict 30-day postoperative complications and mortality, supporting biomarker-driven risk stratification and screening.

Research Themes

  • Biomarker-based perioperative risk stratification
  • Innate immune mechanisms driving perioperative AKI
  • Preoperative metabolic screening and optimization

Selected Articles

1. Soluble urokinase receptor is a kidney-specific vasoconstrictor.

84Level IIICohortEBioMedicine · 2025PMID: 41187619

Integrating a matched cardiac surgery cohort with porcine perfusion and intravital mouse imaging, the authors show that suPAR acts as a kidney-specific vasoconstrictor that reduces renal blood flow and glomerular perfusion. This mechanistic link between innate immune activation and renal hemodynamics provides a plausible causal pathway for perioperative AKI risk.

Impact: First mechanistic evidence that an innate immune mediator directly causes renal vasoconstriction across species and systems, reframing AKI risk beyond tubular injury. It opens biomarker-guided risk stratification and therapeutic targeting of suPAR.

Clinical Implications: Preoperative suPAR measurement may refine AKI risk prediction in cardiac and high-risk surgery; strategies to lower suPAR or blunt its vasoconstrictive signaling could prevent AKI. Anesthesiologists should consider suPAR when evaluating renal hemodynamic vulnerability.

Key Findings

  • Higher suPAR levels were inversely associated with baseline eGFR in a propensity-score–matched cardiac surgery cohort.
  • Ex vivo porcine kidney perfusion with suPAR reduced renal blood flow and increased renal vascular resistance.
  • Intravital multiphoton imaging showed suPAR-induced afferent arteriolar constriction and reduced glomerular perfusion.
  • suPAR is characterized as a predominantly kidney-specific vasoconstrictor with implications for perioperative AKI.

Methodological Strengths

  • Translational, multi-system approach integrating clinical, ex vivo, and in vivo models.
  • Propensity-score–matched clinical cohort strengthens causal inference alongside mechanistic imaging.

Limitations

  • Exact clinical sample size and interventional suPAR-lowering trials are not reported.
  • Generalizability beyond cardiac surgery and species translation requires further validation.

Future Directions: Test suPAR-guided perioperative pathways and evaluate therapies that reduce suPAR levels or block downstream vasoconstrictive signaling to prevent AKI.

2. Association of Liver Fibrosis Fibrosis-4 Score with Perioperative Complications and Mortality: A Retrospective Multicenter Analysis.

70.5Level IIICohortAnesthesiology · 2025PMID: 41191454

In 1,325,102 anesthetized patients, higher FIB-4 categories strongly associated with 30-day mortality and with AKI, myocardial injury, and pulmonary complications, with dose-response effects and robustness to age-adjusted cutoffs. This supports integrating FIB-4 into preoperative risk assessment, even in patients without known liver disease.

Impact: Massive, multicenter analysis demonstrates that a simple, readily available fibrosis score independently stratifies perioperative risk across outcomes, enabling scalable preoperative optimization.

Clinical Implications: Calculate FIB-4 during preoperative evaluation to refine discussions about 30-day mortality and complications and prioritize optimization (e.g., metabolic control, hemodynamic vigilance) for higher-risk categories.

Key Findings

  • Among 1,325,102 patients, FIB-4 1.3–2.67 was associated with cOR 1.533 for 30-day mortality; FIB-4 ≥2.67 with cOR 3.765.
  • Dose–response relationship between continuous FIB-4 and mortality; associations persisted with age-adjusted cutoffs.
  • Elevated FIB-4 predicted AKI (cOR 1.515), myocardial injury (cOR 1.657), and pulmonary complications (cOR 1.323).

Methodological Strengths

  • Extraordinarily large, multicenter cohort with mixed-effects multivariable adjustment.
  • Robustness checks including age-adjusted FIB-4 thresholds and continuous modeling.

Limitations

  • Retrospective design with potential residual confounding.
  • FIB-4 depends on laboratory availability and may be missing in some preoperative workflows.

Future Directions: Prospective validation and interventional studies testing FIB-4–guided optimization pathways and thresholds for perioperative management.

3. Preoperative Hemoglobin A1C, Glycemic Status, and Postoperative Outcomes in General Surgery.

69Level IIICohortJAMA surgery · 2025PMID: 41191374

Using NSQIP data from 282,131 adults, both diagnosed and undiagnosed dysglycemia were common and independently associated with increased 30-day complications and mortality. Risk rose progressively with higher HbA1c, supporting routine preoperative HbA1c screening and tailored glycemic management.

Impact: Defines the perioperative risk gradient across the HbA1c spectrum and quantifies risks in undiagnosed diabetes at scale, informing screening policies.

Clinical Implications: Implement routine preoperative HbA1c testing to uncover undiagnosed diabetes and guide perioperative glucose targets and monitoring intensity.

Key Findings

  • Among 282,131 patients, 36% had diagnosed diabetes and 6.4% had diabetes-range HbA1c without diagnosis.
  • Complication risk increased progressively with higher HbA1c; HbA1c >9.0% associated with OR 1.32 for any complication.
  • Undiagnosed diabetes was associated with higher medical complications (OR 1.11) and mortality (OR 1.24) within 30 days.

Methodological Strengths

  • Very large, multicenter clinical dataset with multivariable adjustment.
  • Clear stratification by diagnosed vs undiagnosed diabetes and graded HbA1c levels.

Limitations

  • Retrospective design with potential selection bias in who had HbA1c measured.
  • Lack of randomized glycemic management limits causal inference on treatment effects.

Future Directions: Prospective trials to test HbA1c-guided perioperative management algorithms and thresholds for intervention.